Reham William Naguib Doss

Reham William Naguib Doss

Beni-Suef University

H-index: 6

Africa-Egypt

About Reham William Naguib Doss

Reham William Naguib Doss, With an exceptional h-index of 6 and a recent h-index of 5 (since 2020), a distinguished researcher at Beni-Suef University, specializes in the field of Dermatology.

His recent articles reflect a diverse array of research interests and contributions to the field:

Level of long noncoding RNA taurine upregulated gene 1 after narrow band-ultraviolet B phototherapy in vitiligo

MicroRNA 155 in lichen planus

Long noncoding RNA (taurine upregulated gene 1) and micro RNA-377: emerging players in the development of metabolic syndrome among psoriasis patients

SOCS3 expression in cutaneous lichen planus

YKL-40 tissue expression in lichen planus

Tissue and serum lipoxin A4 levels in psoriasis patients and their relation with disease severity

Expression of long noncoding RNA in skin exosomes of patients with vitiligo

Cutaneous syndecan-1 expression before and after phototherapy in psoriasis

Reham William Naguib Doss Information

University

Beni-Suef University

Position

lecturer dermatologist Faculty of Medicine Egypt

Citations(all)

128

Citations(since 2020)

93

Cited By

72

hIndex(all)

6

hIndex(since 2020)

5

i10Index(all)

6

i10Index(since 2020)

4

Email

University Profile Page

Beni-Suef University

Reham William Naguib Doss Skills & Research Interests

Dermatology

Top articles of Reham William Naguib Doss

Level of long noncoding RNA taurine upregulated gene 1 after narrow band-ultraviolet B phototherapy in vitiligo

Authors

Reham W Doss,Laila A Rashed,Hanaa Ibrahim,Rehab M Naguib

Journal

Journal of the Egyptian Women’s Dermatologic Society

Published Date

2024/5/1

BackgroundVitiligo is a skin disease that is characterized by depigmenting lesions as a result of melanocytes destruction. Long noncoding Taurine upregulated gene 1 (lnc TUG-1) is one of the long noncoding RNA (lnc RNA) family members. The role of lncRNA TUG1 in melanogenesis has been investigated in recent studies.ObjectiveTo investigate the impact of narrow-band ultraviolet B (NB-UVB) on lncTUG-1 expression in vitiligo.

MicroRNA 155 in lichen planus

Authors

Reham W Doss,Laila A Rashed,Rehab M Naguib

Journal

Journal of the Egyptian Women’s Dermatologic Society

Published Date

2023/9/1

Lichen planus (LP) is a chronic idiopathic inflammatory papulosquamous disease. Tumor necrosis factor-α, interferon (IFN)-γ, interleukin (IL)-2, IL-6, and IL-17 are among the cytokines involved in LP autoimmune pathogenesis [1].

Long noncoding RNA (taurine upregulated gene 1) and micro RNA-377: emerging players in the development of metabolic syndrome among psoriasis patients

Authors

Maha Alhelf,Laila Rashed,RW Doss,Sahar M Mohamed,Naglaa Adly) Abd Elazeem

Journal

Beni-Suef University Journal of Basic and Applied Sciences

Published Date

2023/7/19

BackgroundPsoriasis (PsO) is an immune-mediated dermatosis and systemic inflammatory condition that can affect the skin, joints, and other organs and tissues with a range of comorbidities. The activation of proinflammatory cytokines is the primary cause of the development of skin lesions in PsO. Patients with PsO have a higher risk of developing cardiovascular metabolic comorbidities; among these is the metabolic syndrome (MetS). Particularly, MetS is characterized by abdominal obesity, hypertension, hyperglycemia, and hyperlipidemia, has been linked to PsO. The connection between PsO and MetS is believed to be caused by PsO generating systemic inflammation, which then results in elevated inflammatory adipokines, endothelial dysfunction, and insulin resistance. Micro RNA-377 and long noncoding RNA taurine upregulated 1 (TUG1) are both involved in the control of a variety of inflammatory disorders …

SOCS3 expression in cutaneous lichen planus

Authors

Reham William Doss,Abdel-Aziz El-Rifaie,Laila Ahmed Rashed,Rehab Naguib

Published Date

2023/4/10

Background: Lichen Planus (LP) is an idiopathic, inflammatory, autoimmune disease. The aetiopathogenesis of LP is still unclear. Suppressor of cytokine signaling-3 (SOCS3) is known to inhibit cytokine signaling through Janus kinase (JAK)/signal transducers, activators of transcription, NFkB, and focal adhesion kinase (FAK) signaling pathways. Modulation of SOCS3 level leads to immune dysfunction.Aim of the work: Our study aimed to detect a possible role of SOCS3 in cutaneous LPMethods: In this case-control study skin biopsies were collected from 30 LP patients and 30 healthy volunteers. Biopsies were kept in lysis solution frozen at-80 Celsius till analysis of SOCS3 using Real time polymerase chain technique (RT-PCR) was done.Results: The expression of SOCS3 in cutaneous LP was significantly higher when compared to normal skin from healthy controls (p< 0.001).Conclusion: SOCS3 might play a role in the pathogenesis of LPSuppressor of cytokine signaling-3SOCS3Lichen planusqRT PCRFiguresFigure 1Figure 1IntroductionThe suppressors of cytokine signaling (SOCS) are inhibitors of cytokine signaling that function via the Janus kinase/signal transducers and activators of transcription (JAK/STAT) pathways. 1

YKL-40 tissue expression in lichen planus

Authors

Reham W Doss,Laila A Rashed,Mona EA Abdellatif

Journal

Journal of the Egyptian Women’s Dermatologic Society

Published Date

2022/9/1

BackgroundLichen planus (LP) is an idiopathic chronic inflammatory disease of the skin, mucous membranes, and nails. The exact pathogenesis of LP is still unclear. Chitinase-3-like protein 1 (YKL-40) is one of the 18 glycosyl hydrolases, but lacks enzymatic activity. Recent studies have shown that YKL-40 is present at sites of inflammation and tissue remodeling, triggers immune responses, and attracts eosinophils and T cells.Objective

Tissue and serum lipoxin A4 levels in psoriasis patients and their relation with disease severity

Authors

Rehab Mohamed Naguib,Hanan Abo Zaid Mohamed Al Nemr,Laila Ahmed Rashed,Reham William Naguib

Journal

Egyptian Journal of Dermatology and Venerology

Published Date

2022/5/1

BackgroundPsoriasis disease is a chronic hyperproliferative inflammatory disease, in which genetic and environmental factors have an important role, but the exact cause is yet unknown. Lipoxin A4 plays a role in improvement of psoriasis cases, it blocks the production of the proinflammatory cytokine tumor necrosis factor with increasing the production of the anti-inflammatory cytokine, c–c chemochine receptor-5 by T lymphocytes, and it reduces the perception of pain due to inflammation.AimDetection of the expression of lipoxin A4 in the tissue and serum of psoriatic patients as compared with normal-control persons to investigate the possible role of lipoxin A4 in the psoriasis pathogenesis and its correlation with disease severity.

Expression of long noncoding RNA in skin exosomes of patients with vitiligo

Authors

Reham William Doss,Abdel Aziz Elrifaie,Nancy Magdy Mamdouh,Dina Sabry

Journal

Journal of the Egyptian Women’s Dermatologic Society

Published Date

2020/9/1

BackgroundVitiligo is a relatively common pigmentation disorder in which the skin melanocytes are lost or destroyed. Experimental downregulation of long noncoding RNA H19 (lnc RNA H19) in keratinocytes promoted melanocytes melanogenesis. lnc RNA H19 acts as an important regulator for micro-RNA let7a (miRNA let7a). miRNA let7a has a powerful role in regulation of cell survival and inducing cell apoptosis.ObjectivesThis study was performed to clarify the suggested role of lnc RNA H19 and miRNA let7a derived from human skin exosomes in vitiligo pathogenesis.

Cutaneous syndecan-1 expression before and after phototherapy in psoriasis

Authors

Reham William Doss,Abdel-Aziz El-Rifaie,Aya Nagy Said,Laila A Rashed

Journal

Indian Journal of Dermatology, Venereology and Leprology

Published Date

2020/7/1

Syndecans are transmembrane proteins that comprise a major family of cell surface heparan sulfate proteoglycans. Syndecan‑1, present on the surface of keratinocytes, has the ability to interact with other cell surface receptors and to regulate their actions, controlling cell adhesion, migration and proliferation. 1The expression of syndecan is markedly diminished in diseases that exhibit acantholysis, spongiosis or loss of cellular adhesion such as pemphigus vulgaris, bullous pemphigoid and herpes simplex. 1 Its serum levels has also been used as a marker of severity of inflammation in Crohns disease. 2

See List of Professors in Reham William Naguib Doss University(Beni-Suef University)

Reham William Naguib Doss FAQs

What is Reham William Naguib Doss's h-index at Beni-Suef University?

The h-index of Reham William Naguib Doss has been 5 since 2020 and 6 in total.

What are Reham William Naguib Doss's top articles?

The articles with the titles of

Level of long noncoding RNA taurine upregulated gene 1 after narrow band-ultraviolet B phototherapy in vitiligo

MicroRNA 155 in lichen planus

Long noncoding RNA (taurine upregulated gene 1) and micro RNA-377: emerging players in the development of metabolic syndrome among psoriasis patients

SOCS3 expression in cutaneous lichen planus

YKL-40 tissue expression in lichen planus

Tissue and serum lipoxin A4 levels in psoriasis patients and their relation with disease severity

Expression of long noncoding RNA in skin exosomes of patients with vitiligo

Cutaneous syndecan-1 expression before and after phototherapy in psoriasis

are the top articles of Reham William Naguib Doss at Beni-Suef University.

What are Reham William Naguib Doss's research interests?

The research interests of Reham William Naguib Doss are: Dermatology

What is Reham William Naguib Doss's total number of citations?

Reham William Naguib Doss has 128 citations in total.

What are the co-authors of Reham William Naguib Doss?

The co-authors of Reham William Naguib Doss are Olfat Shaker, Amira El Tawdy.

Co-Authors

H-index: 46
Olfat Shaker

Olfat Shaker

Cairo University

H-index: 16
Amira El Tawdy

Amira El Tawdy

Cairo University

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