Plamena R. Angelova

About Plamena R. Angelova

Plamena R. Angelova, With an exceptional h-index of 34 and a recent h-index of 31 (since 2020), a distinguished researcher at University College London,

His recent articles reflect a diverse array of research interests and contributions to the field:

Mitochondrial Permeability Transition, Cell Death and Neurodegeneration

High levels of FAD autofluorescence indicate pathology preceding cell death

Astrocytes produce nitric oxide via nitrite reduction in mitochondria to regulate cerebral blood flow during brain hypoxia

Nrf2 depletion in the context of loss-of-function Keap1 leads to mitolysosome accumulation

Carbon monoxide neurotoxicity is triggered by oxidative stress induced by ROS production from three distinct cellular sources

Correction: Current advances in gene therapy of mitochondrial diseases

Protein aggregation and calcium dysregulation are hallmarks of familial Parkinson’s disease in midbrain dopaminergic neurons

P2X7 receptor and purinergic signaling: orchestrating mitochondrial dysfunction in neurodegenerative diseases

Plamena R. Angelova Information

University

Position

___

Citations(all)

5772

Citations(since 2020)

4448

Cited By

2877

hIndex(all)

34

hIndex(since 2020)

31

i10Index(all)

52

i10Index(since 2020)

49

Email

University Profile Page

Google Scholar

Top articles of Plamena R. Angelova

Mitochondrial Permeability Transition, Cell Death and Neurodegeneration

2024/4/8

High levels of FAD autofluorescence indicate pathology preceding cell death

Biochimica et Biophysica Acta (BBA)-General Subjects

2024/1/1

Astrocytes produce nitric oxide via nitrite reduction in mitochondria to regulate cerebral blood flow during brain hypoxia

Cell Reports

2023/12/26

Nrf2 depletion in the context of loss-of-function Keap1 leads to mitolysosome accumulation

Free Radical Biology and Medicine

2023/11/1

Carbon monoxide neurotoxicity is triggered by oxidative stress induced by ROS production from three distinct cellular sources

Redox Biology

2023/4/1

Correction: Current advances in gene therapy of mitochondrial diseases

2023/2/8

Protein aggregation and calcium dysregulation are hallmarks of familial Parkinson’s disease in midbrain dopaminergic neurons

npj Parkinson's Disease

2022/11/24

P2X7 receptor and purinergic signaling: orchestrating mitochondrial dysfunction in neurodegenerative diseases

2022/11/1

Pathological structural conversion of α-synuclein at the mitochondria induces neuronal toxicity

Nature neuroscience

2022/9

Hyperammonaemia induces mitochondrial dysfunction and neuronal cell death

JHEP Reports

2022/8/1

Pharmacological sequestration of mitochondrial calcium uptake protects against dementia and β-amyloid neurotoxicity

Scientific Reports

2022/7/27

Deuterated arachidonic acid ameliorates lipopolysaccharide-induced lung damage in mice

Antioxidants

2022/3/31

Astrocyte mitochondria produce nitric oxide from nitrite to modulate cerebral blood flow during brain hypoxia

BioRxiv

2022/3/14

Age‐related changes in the energy of human mesenchymal stem cells

Journal of cellular physiology

2022/3

Probing cell redox state and glutathione-modulating factors using a monochlorobimane-based microplate assay

Antioxidants

2022/2/15

Metabolically induced intracellular pH changes activate mitophagy, autophagy, and cell protection in familial forms of Parkinson's disease

The FEBS Journal

2022/2

Variability of mitochondrial energy balance across brain regions

Journal of Neurochemistry

2021/5

Phospholipase iPLA2β averts ferroptosis by eliminating a redox lipid death signal

Nature chemical biology

2021/4

Viper toxins affect membrane characteristics of human erythrocytes

Biophysical Chemistry

2021/3/1

See List of Professors in Plamena R. Angelova University(University College London)