A Watson

A Watson

Monash University

H-index: 28

Oceania-Australia

About A Watson

A Watson, With an exceptional h-index of 28 and a recent h-index of 19 (since 2020), a distinguished researcher at Monash University,

His recent articles reflect a diverse array of research interests and contributions to the field:

Cardiovascular characterisation of a novel mouse model that combines hypertension and diabetes co-morbidities

Endothelial reactive oxygen-forming NADPH oxidase 5 is a possible player in diabetic aortic aneurysm but not atherosclerosis

Designing publicly funded organisations for accelerated low carbon innovation: A case study of the ETI, UK and ARPA-E, US

The role of activator protein-1 (AP-1) transcription factors in diabetes associated atherosclerosis

Role of cFOS in mechanosensitive transcriptional regulation in diabetes associated atherosclerosis

Transcription Factor cFOS Mediated Foam Cell Formation in Diabetes Associated Atherosclerosis

Corrigendum: Characterising an Alternative Murine Model of Diabetic Cardiomyopathy

Renal deafferentation prevents progression of hypertension and changes to sympathetic reflexes in a rabbit model of chronic kidney disease

A Watson Information

University

Monash University

Position

___

Citations(all)

3463

Citations(since 2020)

1024

Cited By

2834

hIndex(all)

28

hIndex(since 2020)

19

i10Index(all)

36

i10Index(since 2020)

25

Email

University Profile Page

Monash University

Top articles of A Watson

Cardiovascular characterisation of a novel mouse model that combines hypertension and diabetes co-morbidities

Authors

Arpeeta Sharma,Judy SY Choi,Anna MD Watson,Leila Li,Thomas Sonntag,Man KS Lee,Andrew J Murphy,Miles De Blasio,Geoffrey A Head,Rebecca H Ritchie,Judy B de Haan

Journal

Scientific Reports

Published Date

2023/5/30

Epidemiologic data suggest that the prevalence of hypertension in patients with diabetes mellitus is ∼1.5–2.0 times greater than in matched non-diabetic patients. This co-existent disease burden exacerbates cardiac and vascular injury, leading to structural and functional changes to the myocardium, impaired cardiac function and heart failure. Oxidative stress and persistent low-grade inflammation underlie both conditions, and are identified as major contributors to pathological cardiac remodelling. There is an urgent need for effective therapies that specifically target oxidative stress and inflammation to protect against cardiac remodelling. Animal models are a valuable tool for testing emerging therapeutics, however, there is a notable lack of appropriate animal models of co-morbid diabetes and hypertension. In this study, we describe a novel preclinical mouse model combining diabetes and hypertension to …

Endothelial reactive oxygen-forming NADPH oxidase 5 is a possible player in diabetic aortic aneurysm but not atherosclerosis

Authors

Florence Ho,Anna MD Watson,Mahmoud H Elbatreek,Pamela WM Kleikers,Waheed Khan,Karly C Sourris,Aozhi Dai,Jay Jha,Harald HHW Schmidt,Karin AM Jandeleit-Dahm

Journal

Scientific Reports

Published Date

2022/7/7

Atherosclerosis and its complications are major causes of cardiovascular morbidity and death. Apart from risk factors such as hypercholesterolemia and inflammation, the causal molecular mechanisms are unknown. One proposed causal mechanism involves elevated levels of reactive oxygen species (ROS). Indeed, early expression of the ROS forming NADPH oxidase type 5 (Nox5) in vascular endothelial cells correlates with atherosclerosis and aortic aneurysm. Here we test the pro-atherogenic Nox5 hypothesis using mouse models. Because Nox5 is missing from the mouse genome, a knock-in mouse model expressing human Nox5 in its physiological location of endothelial cells (eNOX5ki/ki) was tested as a possible new humanised mouse atherosclerosis model. However, whether just on a high cholesterol diet or by crossing in aortic atherosclerosis-prone ApoE−/− mice with and without induction of diabetes …

Designing publicly funded organisations for accelerated low carbon innovation: A case study of the ETI, UK and ARPA-E, US

Authors

Anna Watson

Journal

Energy Policy

Published Date

2022/9/1

The role of publicly funded organisations in implementing accelerated low carbon innovation policy is receiving increasing attention. Due to different national contexts and priorities however, policy makers face high levels of complexity in understanding what organisational approaches might best suit their aims. This paper develops a set of ten principles for accelerated innovation organisation design, which seek to provide policy makers with a tool to better understand the interplay of different design features on innovation outputs. The principles are applied to a comparative case study of the Energy Technologies Institute (ETI), United Kingdom (UK), and the Advanced Research Projects Agency– Energy (ARPA-E), United States (US). Results demonstrate that the design of the ETI, which embedded incumbent industry actors and engaged a narrower set of staff competencies, caused it to prioritise un transformative …

The role of activator protein-1 (AP-1) transcription factors in diabetes associated atherosclerosis

Authors

W Khan,MKS Lee,AM Watson,S Maxwell,ME Cooper,A Murphy,S Baratchi,KA Jandeleit-Dahm

Journal

Atherosclerosis

Published Date

2022/8/1

Methods: To address this, we applied single cell RNA sequencing (scRNA-seq) to identify cell specific transcriptional changes of the diabetic aorta. Further mechanistic experiments were performed in human aortic endothelial cells (HAECs) and THP-1-derived macrophages. For scRNA-seq, viable cells from diabetic murine aorta (10 weeks post streptozotocin induced diabetes in Apoe-/-mice) were subjected to scRNA-seq library preparation (10X Genomics) and Illumina Nova-seq 6000.Results: Graph-based clustering identified several cellular clusters including endothelial cells, macrophages, and foam cells. Pathway analysis identified cell specific pathways such as cholesterol metabolism and oxidative phosphorylation in foam cells and “flow shear stress and atherosclerosis” in endothelial cells. AP-1 transcription factors were most significantly dysregulated genes in these pathways. In vitro experiments …

Role of cFOS in mechanosensitive transcriptional regulation in diabetes associated atherosclerosis

Authors

A Khan,M Lee,A Watson,S Maxwell,M Cooper,A Murphy,K Jandeleit-Dahm

Journal

European Heart Journal

Published Date

2021/10/1

Atherosclerosis, as manifested clinically by myocardial infarction, stroke and peripheral vascular disease, is a major contributor to cardiovascular disease, the leading cause of death in patients with diabetes. The lipid-laden plaque development within the arterial vessel wall is a progressive process initiated with endothelial cell activation and monocyte adhesion. These cellular events occur primarily at the regions of blood vessels exposed to turbulent blood flow (TBF) and low shear stress such as vascular bends and bifurcations. Exposure of the vascular cells to chronic hyperglycaemia and TBF induces a proatherogenic transcriptional profile. Studies have shown that shear stress regulates vascular pathophysiology via differential regulation of transcription factors (TFs) such as KLF4, EGR1 and AP-1, hence named as mechanosensitive TFs. AP-1 is a heterodimer composed of FOS, Jun and ATF family of TFs …

Transcription Factor cFOS Mediated Foam Cell Formation in Diabetes Associated Atherosclerosis

Authors

A Khan,M Lee,A Watson,S Maxwell,M Cooper,A Murphy,K Jandeleit-Dahm

Journal

Heart, Lung and Circulation

Published Date

2021/1/1

BackgroundAtherosclerotic cardiovascular disease is a leading cause of death in patients with diabetes. Endothelial activation, monocytes cross-endothelial migration and engulfment of oxidized low-density lipoprotein (ox-LDL) by macrophages to transform into foam cells are key steps in the atherosclerotic plaque development. Recent single cell transcriptomic (SCT) analysis of aortic leukocytes identified three distinct macrophage populations including foam cells with higher expression of Trem2 in western diet fed murine models of atherosclerosis. Hyperglycaemic environment due to diabetes is known to affect cellular phenotypes, however the molecular mechanisms responsible for diabetes-induced foam cell formation in atherosclerosis are not known.MethodsWe applied SCT approach to identify cell specific transcriptomic landscape of the diabetic aorta. SCT data was complemented with In vitro experiments …

Corrigendum: Characterising an Alternative Murine Model of Diabetic Cardiomyopathy

Authors

Mitchel Tate,Darnel Prakoso,Andrew M Willis,Cheng Peng,Minh Deo,Cheng Xue Qin,Jesse L Walsh,David M Nash,Charles D Cohen,Alex K Rofe,Arpeeta Sharma,Helen Kiriazis,Daniel G Donner,Judy B De Haan,Anna MD Watson,Miles J De Blasio,Rebecca H Ritchie

Journal

Frontiers in Physiology

Published Date

2021/8/19

1 Heart Failure Pharmacology, Baker Heart and Diabetes Institute, Melbourne, VIC, Australia, 2 Department of Diabetes, Central Clinical School, Monash University, Melbourne, VIC, Australia, 3 School of Biosciences, The University of Melbourne, Melbourne, VIC, Australia, 4 Oxidative Stress Laboratory, Baker Heart and Diabetes Institute, Melbourne, VIC, Australia, 5 Preclinical Cardiology, Microsurgery and Imaging Platform, Baker Heart and Diabetes Institute, Melbourne, VIC, Australia, 6 Department of Pharmacology and Therapeutics, The University of Melbourne, Melbourne, VIC, Australia

Renal deafferentation prevents progression of hypertension and changes to sympathetic reflexes in a rabbit model of chronic kidney disease

Authors

Yusuke Sata,Sandra L Burke,Nina Eikelis,Anna MD Watson,Cindy Gueguen,Kristy L Jackson,Gavin W Lambert,Kyungjoon Lim,Kate M Denton,Markus P Schlaich,Geoffrey A Head

Journal

Hypertension

Published Date

2021/11

There is increasing evidence that renal denervation is effective in alleviating hypertension associated with elevation of renal sympathetic nerve activity (RSNA) in chronic kidney disease (CKD), but whether this is due to reduction in renal afferent signaling is unclear. We determined the cardiovascular and sympathetic effects of total renal denervation or afferent renal denervation (topical capsaicin) on CKD induced by glomerular layer lesioning of the left kidney and right nephrectomy in conscious rabbits. CKD increased blood pressure by 18±2 mmHg and plasma creatinine by 40% over 2 to 4 weeks (both P<0.001), while RSNA (43%) and total norepinephrine spillover (28%) were elevated in CKD compared with sham (both P=0.04). After total or afferent renal denervation blood pressure, RSNA and norepinephrine spillover were similar or lower than non-CKD (sham) rabbits. While plasma creatinine in CKD rabbits …

Targeted deletion of nicotinamide adenine dinucleotide phosphate oxidase 4 from proximal tubules is dispensable for diabetic kidney disease development

Authors

Vicki Thallas-Bonke,Sih Min Tan,Runa S Lindblom,Matthew Snelson,Cesare Granata,Jay Chandra Jha,Karly C Sourris,Adrienne Laskowski,Anna Watson,Michel Tauc,Isabelle Rubera,Guoping Zheng,Ajay M Shah,David CH Harris,Mahmoud H Elbatreek,Phillip Kantharidis,Mark E Cooper,Karin Jandeleit-Dahm,Melinda T Coughlan

Journal

Nephrology Dialysis Transplantation

Published Date

2021/6/1

Background The nicotinamide adenine dinucleotide phosphate oxidase isoform 4 (Nox4) mediates reactive oxygen species (ROS) production and renal fibrosis in diabetic kidney disease (DKD) at the level of the podocyte. However, the mitochondrial localization of Nox4 and its role as a mitochondrial bioenergetic sensor has recently been reported. Whether Nox4 drives pathology in DKD within the proximal tubular compartment, which is densely packed with mitochondria, is not yet known. Methods We generated a proximal tubular–specific Nox4 knockout mouse model by breeding Nox4flox/flox mice with mice expressing Cre recombinase under the control of the sodium–glucose cotransporter-2 promoter. Subsets of Nox4ptKO mice and their Nox4flox/flox littermates were injected with streptozotocin (STZ) to induce diabetes. Mice were followed for 20 weeks and renal injury …

Inhibitors of advanced glycation end product (AGE) formation and accumulation

Authors

Karly C Sourris,Anna Watson,Karin Jandeleit-Dahm

Journal

Reactive Oxygen Species: Network Pharmacology and Therapeutic Applications

Published Date

2021

A range of chemically different compounds are known to inhibit the formation and accumulation of advanced glycation end products (AGEs) or disrupt associated signalling pathways. There is evidence that some of these agents can provide end-organ protection in chronic diseases including diabetes. Whilst this group of therapeutics are structurally and functionally different and have a range of mechanisms of action, they ultimately reduce the deleterious actions and the tissue burden of advanced glycation end products. To date it remains unclear if this is due to the reduction in tissue AGE levels per se or the modulation of downstream signal pathways. Some of these agents either stimulate antioxidant defence or reduce the formation of reactive oxygen species (ROS), modify lipid profiles and inhibit inflammation. A number of existing treatments for glucose lowering, hypertension and hyperlipidaemia are …

MODULATION OF SYMPATHETIC NERVE ACTIVITY BY SGLT2 INHIBITOR EMPAGLIFLOZIN IN DIABETIC RABBITS

Authors

Cindy Gueguen,Sandra L Burke,Benjamin Barzel,Kyungjoon Lim,Nina Eikelis,Anna Md Watson,Jay C Jha,Kristy L Jackson,Yusuke Sata,Gavin W Lambert,Karin Am Jandeleit-Dahm,Mark E Cooper,Merlin C Thomas,Geoffrey A Head

Journal

Journal of Hypertension

Published Date

2021/4/1

Objective:Treatment with the sodium-glucose cotransporter 2 (SGLT2) inhibitor empagliflozin in patients with type-2 diabetes is associated with 38% reduction in cardiovascular mortality. We explored the notion that the mechanisms underlying these benefits may involve an action on the sympathetic nervous system (SNS).Design and method:Diabetes was induced by alloxan in New-Zealand White rabbits. We determined the associated changes in blood pressure (BP), heart rate (HR) and renal sympathetic nerve activity (RSNA) as well as baroreflex responses to changes in BP and norepinephrine spillover in conscious rabbits. The effects of one-week treatment with empagliflozin were compared to those of another blood glucose-lowering treatment insulin, the proximally-acting natriuretic acetazolamide, the ACE inhibitor perindopril, vehicle treatment and to an untreated diabetic group.Results:Diabetic rabbits were …

Role of FOS transcription factor in diabetes associated atherosclerosis; insight from single cell RNA sequencing

Authors

AW Khan,MSK Lee,AM Watson,S Maxwell,ME Cooper,A Murphy,KA Jandeleit-Dahm

Journal

Atherosclerosis

Published Date

2021/8/1

Methods: For scRNA-seq, diabetes was induced with streptozotocin in Apoe-/-mice and followed for 10 weeks. Cells from digested aortae were subjected to scRNA-seq library preparation (10X Genomics) and sequenced with Illumina Nova-seq 6000.Results: ScRNA-seq unbiasedly uncovered cell specific transcriptional landscape of the atherosclerotic aorta in diabetes. Graph based clustering using Partek flow identified thirteen cellular clusters including macrophages. Trem2 hi macrophages were identified with specific gene markers (Trem2, Cd9, Ctsd) with dysregulated pathways including cholesterol metabolism and oxidative phosphorylation. AP-1 transcription factor (TF) Fos was upregulated only in Trem2 hi macrophages as compared to all other cell types identified in scRNA-seq data. Oil red O staining of THP-1-derived macrophages stimulated with high glucose and ox-LDL showed increased foam cell …

Accelerating Low Carbon Innovation: National Institutions and Effective Organisation Design

Authors

Anna Watson

Published Date

2021

Accelerated clean electricity innovation is essential if global climate change targets are to be met by 2050. National innovation policy strategies are identified as key in delivering this, requiring policy makers to develop new approaches to technology funding and market development. At present, the role of politics in the emergence and implementation of innovation policy is not well understood by the innovation systems literature. There is therefore a gap in knowledge in relation to how national institutions shape the development of clean electricity innovation systems and how publicly funded organisations can be designed to accelerate innovation within them. This thesis addresses this gap by analysing how national institutions have affected the design of two publicly funded organisations for accelerated clean electricity innovation-the Energy Technologies Institute (ETI) in the UK and Advanced Research Projects Agency-Energy (ARPA-E) in the US.The thesis opens with a paper that refines a layered institutional framework, demonstrating the effect of multiple institutions on the development of the UK’s clean electricity innovation system 2000-2018. The second paper builds on this system overview to introduce the case study of the ETI. The ETI’s operation is analysed through a refined framework of ten principles for accelerated innovation organisation design, which explore how the formation and operation of an organisation is affected by the broader institutional conditions introduced in paper one. Paper three further develops the ten principles via a comparative case study of the ETI and ARPA-E. The impact of different national institutional …

Deficiency of microRNA-181a results in transcriptome-wide cell-specific changes in the kidney and increases blood pressure

Authors

Madeleine R Paterson,Kristy L Jackson,Malathi SI Dona,Gabriella E Farrugia,Bruna Visniauskas,Anna MD Watson,Chad Johnson,Minolfa C Prieto,Roger G Evans,Fadi J Charchar,Alexander R Pinto,Francine Z Marques,Geoffrey A Head

Journal

Hypertension

Published Date

2021/11

MicroRNA miR-181a is downregulated in the kidneys of hypertensive patients and hypertensive mice. In vitro, miR-181a is a posttranslational inhibitor of renin expression, but pleiotropic mechanisms by which miR-181a may influence blood pressure (BP) are unknown. Here, we determined whether deletion of miR-181a/b-1 in vivo changes BP and the molecular mechanisms involved at the single-cell level. We developed a KO (knockout) mouse model lacking miR-181a/b-1 genes using CRISPR/Cas9 technology. Radiotelemetry probes were implanted in 12-week-old C57BL/6J WT (wild type) and miR-181a/b-1 KO mice. Systolic and diastolic BP were 4- to 5-mm Hg higher in KO compared with WT mice over 24 hours (P<0.01). Compared with WT mice, renal renin was higher in the juxtaglomerular cells of KO mice. BP was similar in WT mice on a high- (3.1%) versus low- (0.3%) sodium diet (+0.4±0.8 mm Hg …

Designing low carbon innovation organisations: the Energy Technologies Institute experience

Authors

Anna Watson

Journal

Environmental Innovation and Societal Transitions

Published Date

2021/6/1

If clean energy innovation is to be accelerated at the pace required, new approaches to designing public innovation organisations need to be developed. Despite the importance of these organisations in driving sustainability transitions, their role has not yet been explored in the literature. This paper addresses this gap by refining ten principles for publicly funded innovation organisation design and applying them to a detailed case study of the Energy Technologies Institute (ETI), UK. Results demonstrate that the ETI's design reflected broader political and industrial relationships at the time of its formation, embedding six large, incumbent energy firms. This effected its ability to pursue the riskier aspects of its mission, resulting in the pursuit of more incremental technologies and outputs. Generalisable aspects of effective innovation organisation design and opportunities to further develop the principles are also identified.

590 Single Cell Transcriptomic Landscape of Atheroprone Endothelium of Aorta in Diabetes associated Atherosclerosis

Authors

A Khan,M Lee,A Watson,S Maxwell,M Cooper,A Murphy,K Jandeleit-Dahm

Journal

Heart, Lung and Circulation

Published Date

2020/1/1

Cardiovascular disease, a leading cause of death worldwide, is caused mainly by atherosclerosis. Fluid mechanical forces generated by disturbed blood flow (DBF) are long known to cause structural, functional and transcriptional changes in the vascular endothelium and atherosclerosis is known to develop near branches and bends of arteries that are exposed to this DBF. Diabetes accelerates atherosclerosis development and represents an independent risk factor. However, the transcriptional signature of atheroprone endothelium in the diabetic aorta has not been investigated previously. Single cell RNA sequencing (scRNA-seq) has provided a unique opportunity to unbiasedly discover cellular heterogeneity. We applied a scRNA-seq approach to identify the transcriptional signature of atherosusceptible endothelium in diabetes associated atherosclerosis.

Differential sympathetic response to lesion-induced chronic kidney disease in rabbits

Authors

Yusuke Sata,Sandra L Burke,Anna MD Watson,Jay C Jha,Cindy Gueguen,Nina Eikelis,Kyungjoon Lim,Kristy L Jackson,Gavin W Lambert,Karin AM Jandeleit-Dahm,Kate M Denton,Murray D Esler,Markus P Schlaich,Geoffrey A Head

Journal

Kidney International

Published Date

2020/10/1

Chronic kidney disease (CKD) is associated with greater sympathetic nerve activity but it is unclear if this is a kidney-specific response or due to generalized stimulation of sympathetic nervous system activity. To determine this, we used a rabbit model of CKD in which quantitative comparisons with control rabbits could be made of kidney sympathetic nerve activity and whole-body norepinephrine spillover. Rabbits either had surgery to lesion 5/6th of the cortex of one kidney by electro-lesioning and two weeks later removal of the contralateral kidney, or sham lesioning and sham nephrectomy. After three weeks, the blood pressure was statistically significantly 20% higher in conscious rabbits with CKD compared to rabbits with a sham operation, but their heart rate was similar. Strikingly, kidney nerve activity was 37% greater than in controls, with greater burst height and frequency. Total norepinephrine spillover was …

Synergistic Catalysis for Stereodivergent Synthesis of trans-and cis-Skipped Dienes

Authors

Fang Wang,Shenqqing Zhu,Lingling Chu

Journal

Synlett

Published Date

2020/11

Catalytic, stereoselective synthesis of skipped dienes is an important topic in organic synthesis. Summarized here are the transition-metal-catalyzed stereoselective approaches and a new, photoinduced stereodivergent strategy reported by our group recently. Our strategy utilizes a synergistic photoredox/nickel protocol to enable the cross-electrophile coupling of allylic carbonates and vinyl triflates to construct 1,4-dienes, the stereoselectivity of which was tuned by the triplet energy (E T) photocatalysts employed, offering a convenient and stereodivergent solution to (E)- and (Z)-1,4-dienes from one set of substrates.

Empagliflozin modulates renal sympathetic and heart rate baroreflexes in a rabbit model of diabetes

Authors

Cindy Gueguen,Sandra L Burke,Benjamin Barzel,Nina Eikelis,Anna MD Watson,Jay C Jha,Kristy L Jackson,Yusuke Sata,Kyungjoon Lim,Gavin W Lambert,Karin AM Jandeleit-Dahm,Mark E Cooper,Merlin C Thomas,Geoffrey A Head

Journal

Diabetologia

Published Date

2020/7

Aims/hypothesis We determined whether empagliflozin altered renal sympathetic nerve activity (RSNA) and baroreflexes in a diabetes model in conscious rabbits. Methods Diabetes was induced by alloxan, and RSNA, mean arterial pressure (MAP) and heart rate were measured before and after 1 week of treatment with empagliflozin, insulin, the diuretic acetazolamide or the ACE inhibitor perindopril, or no treatment, in conscious rabbits. Results Four weeks after alloxan administration, blood glucose was threefold and MAP 9% higher than non-diabetic controls (p < 0.05). One week of treatment with empagliflozin produced a stable fall in blood glucose (−43%) and increased water intake (+49%) but did not change RSNA, MAP or heart rate compared with untreated diabetic rabbits. The maximum RSNA to hypotension …

Single cell RNA sequencing identifies transcriptional signature of atherosusceptible endothelium of aorta in diabetes associated atherosclerosis

Authors

A Khan,S Lee,A Watson,S Maxwell,M Cooper,A Murphy,K Jandeleit-Dahm

Journal

European Heart Journal

Published Date

2020/11

Background Transcriptomic analyses have provided invaluable information for linking genotypes to phenotypes. However, despite the near identical genotype, each cell types in our body has a unique gene expression signature. Deep sequencing at single cell resolution has provided a unique opportunity to unbiasedly discover cellular heterogeneity, disease associated cell populations and to characterise the cell specific transcriptomic landscape. Cardiovascular (CV) disease, a leading cause of death worldwide, is caused mainly by atherosclerosis, a pathological build-up of plaque within arterial vessel walls. Fluid mechanical forces generated by disturbed blood flow are long known to cause structural and transcriptional changes in the vascular endothelium. Atherosclerosis develops near branches and bends of arteries exposed to disturbed blood flow. Diabetes accelerates atherosclerosis …

See List of Professors in A Watson University(Monash University)

A Watson FAQs

What is A Watson's h-index at Monash University?

The h-index of A Watson has been 19 since 2020 and 28 in total.

What are A Watson's top articles?

The articles with the titles of

Cardiovascular characterisation of a novel mouse model that combines hypertension and diabetes co-morbidities

Endothelial reactive oxygen-forming NADPH oxidase 5 is a possible player in diabetic aortic aneurysm but not atherosclerosis

Designing publicly funded organisations for accelerated low carbon innovation: A case study of the ETI, UK and ARPA-E, US

The role of activator protein-1 (AP-1) transcription factors in diabetes associated atherosclerosis

Role of cFOS in mechanosensitive transcriptional regulation in diabetes associated atherosclerosis

Transcription Factor cFOS Mediated Foam Cell Formation in Diabetes Associated Atherosclerosis

Corrigendum: Characterising an Alternative Murine Model of Diabetic Cardiomyopathy

Renal deafferentation prevents progression of hypertension and changes to sympathetic reflexes in a rabbit model of chronic kidney disease

...

are the top articles of A Watson at Monash University.

What is A Watson's total number of citations?

A Watson has 3,463 citations in total.

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