Ida G Lunde

About Ida G Lunde

Ida G Lunde, With an exceptional h-index of 36 and a recent h-index of 30 (since 2020), a distinguished researcher at Universitetet i Oslo, specializes in the field of Basic science cardiology.

His recent articles reflect a diverse array of research interests and contributions to the field:

Emerging roles for the ADAMTS-like family of matricellular proteins in cardiovascular disease through regulation of the extracellular microenvironment

Regulation of cardiomyocyte t‐tubule structure by preload and afterload: Roles in cardiac compensation and decompensation

Inhibition of the extracellular enzyme A disintegrin and metalloprotease with thrombospondin motif 4 prevents cardiac fibrosis and dysfunction

Stretch harmonizes sarcomere strain across the cardiomyocyte

Beneficial effects of exercise initiated before development of hypertrophic cardiomyopathy in genotype-positive mice

Inflammation and syndecan-4 shedding from cardiac cells in ischemic and non-ischemic heart disease

Prevalence and clinical consequences of multiple pathogenic variants in dilated cardiomyopathy

Lumican accumulates with fibrillar collagen in fibrosis in hypertrophic cardiomyopathy

Ida G Lunde Information

University

Position

Oslo University Hospital

Citations(all)

3932

Citations(since 2020)

2853

Cited By

2163

hIndex(all)

36

hIndex(since 2020)

30

i10Index(all)

48

i10Index(since 2020)

46

Email

University Profile Page

Google Scholar

Ida G Lunde Skills & Research Interests

Basic science cardiology

Top articles of Ida G Lunde

Emerging roles for the ADAMTS-like family of matricellular proteins in cardiovascular disease through regulation of the extracellular microenvironment

2024/12

Ida G Lunde
Ida G Lunde

H-Index: 25

Regulation of cardiomyocyte t‐tubule structure by preload and afterload: Roles in cardiac compensation and decompensation

The Journal of Physiology

2024

Inhibition of the extracellular enzyme A disintegrin and metalloprotease with thrombospondin motif 4 prevents cardiac fibrosis and dysfunction

Cardiovascular Research

2023/8

Stretch harmonizes sarcomere strain across the cardiomyocyte

Circulation Research

2023/7/21

Beneficial effects of exercise initiated before development of hypertrophic cardiomyopathy in genotype-positive mice

American Journal of Physiology-Heart and Circulatory Physiology

2023/5/17

Inflammation and syndecan-4 shedding from cardiac cells in ischemic and non-ischemic heart disease

Biomedicines

2023/4/1

Prevalence and clinical consequences of multiple pathogenic variants in dilated cardiomyopathy

Circulation: Genomic and Precision Medicine

2023/4

Lumican accumulates with fibrillar collagen in fibrosis in hypertrophic cardiomyopathy

ESC Heart Failure

2023/4

Temporal expression and spatial distribution of the proteoglycan versican during cardiac fibrosis development

Matrix Biology Plus

2023/12/1

Atrial Sarcomeric Dysfunction in Ttntv Mice Indicates a Causal Relationship Between Rare Variation in TTN and Early Onset Atrial Fibrillation in Patients

Circulation

2023/11/7

Acute heart failure: mechanisms and pre-clinical models—a Scientific Statement of the ESC Working Group on Myocardial Function

2023/10

ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overload

Communications Biology

2022/12/20

Cardiomyocyte-specific overexpression of syndecan-4 in mice results in activation of calcineurin-NFAT signalling and exacerbated cardiac hypertrophy

Molecular Biology Reports

2022/12

Ida G Lunde
Ida G Lunde

H-Index: 25

Calcineurin-NFAT dynamics correspond to cardiac remodeling during aortic banding and debanding, mimicking aortic valve replacement

Frontiers in Molecular Medicine

2022/10/25

Ida G Lunde
Ida G Lunde

H-Index: 25

Theis Tønnessen
Theis Tønnessen

H-Index: 22

Deletion of the extracellular matrix glycoprotein ADAMTSL3 increases pro-fibrotic signalling and exacerbates heart failure in mice

European Heart Journal

2022/10/1

Titin truncating variant cardiomyopathy and related sarcomere insufficiency causes high energy demand resulting in mitochondrial dysfunction, autophagosome formation, and apoptosis

European Heart Journal

2022/10/1

Exercise training during childhood and adolescence is associated with favorable diastolic function in hypertrophic cardiomyopathy

International Journal of Cardiology

2022/10/1

Ida G Lunde
Ida G Lunde

H-Index: 25

Thor Edvardsen
Thor Edvardsen

H-Index: 65

Abstract P2052: Lowered Pro-fibrotic And Damage Associated Molecular Pattern Gene Expressions After Exercise Training In A Mouse Model Of Hypertrophic Cardiomyopathy

Circulation Research

2022/8/5

Thor Edvardsen
Thor Edvardsen

H-Index: 65

Ida G Lunde
Ida G Lunde

H-Index: 25

Beneficial effects of exercise training on expression of fibrosis-related genes in a mouse model of hypertrophic cardiomyopathy

Cardiovascular Research

2022/6/1

ProANP31-67 ameliorates adverse cardiac remodeling and improves systolic and diastolic functions in a preclinical model of cardiorenal syndrome

Cardiovascular Research

2022/6/1

See List of Professors in Ida G Lunde University(Universitetet i Oslo)