Christian Staehr

The vascular Na, K-ATPase: Clinical implications in stroke, migraine, and hypertension

Neurovascular uncoupling is linked to microcirculatory dysfunction in regions outside the ischemic core following ischemic stroke

Hypercontractile Cardiac Phenotype in Mice with Migraine-Associated Mutation in the Na+,K+-ATPase α2-Isoform

SorCS2 modulates neurovascular coupling via glutamatergic and calcium signaling in astrocytes

Microcirculatory dysfunction associates with neurovascular uncoupling in peri-ischemic brain regions after ischemic stroke

Migraine‐Associated Mutation in the Na, K‐ATPase Leads to Disturbances in Cardiac Metabolism and Reduced Cardiac Function

Stress adaptation in rats associate with reduced expression of cerebrovascular Kv7.4 channels and biphasic neurovascular responses

Demand creates its own supply: The Na/K‐ATPase controls metabolic reserve and flexibility