Takashi Saito

About Takashi Saito

Takashi Saito, With an exceptional h-index of 49 and a recent h-index of 44 (since 2020), a distinguished researcher at Nagoya City University, specializes in the field of Neurodegenerative Disorders.

His recent articles reflect a diverse array of research interests and contributions to the field:

Automatic test object driving device, automatic test object driving method, and test object testing system

Aβ-driven nuclear pore complex dysfunction alters activation of necroptosis proteins in a mouse model of Alzheimer’s Disease

β-amyloid accumulation enhances microtubule associated protein tau pathology in an APPNL-GF/MAPTP301S mouse model of Alzheimer’s disease

Temporal alterations in white matter in an App knock-in mouse model of Alzheimer's disease.

Inflammasome signaling is dispensable for ß-amyloid-induced neuropathology in preclinical models of Alzheimer's disease.

Proteomic alterations in the brain and blood-brain barrier during brain Aβ accumulation in an APP knock-in mouse model of Alzheimer's disease.

A microtubule stabilizer ameliorates protein pathogenesis and neurodegeneration in mouse models of repetitive traumatic brain injury.

Translocator protein is a marker of activated microglia in rodent models but not human neurodegenerative diseases.

Takashi Saito Information

University

Position

___

Citations(all)

10077

Citations(since 2020)

7596

Cited By

4777

hIndex(all)

49

hIndex(since 2020)

44

i10Index(all)

109

i10Index(since 2020)

105

Email

University Profile Page

Google Scholar

Takashi Saito Skills & Research Interests

Neurodegenerative Disorders

Top articles of Takashi Saito

Automatic test object driving device, automatic test object driving method, and test object testing system

2024/1/9

Aβ-driven nuclear pore complex dysfunction alters activation of necroptosis proteins in a mouse model of Alzheimer’s Disease

bioRxiv

2024/1/9

Takashi Saito
Takashi Saito

H-Index: 31

β-amyloid accumulation enhances microtubule associated protein tau pathology in an APPNL-GF/MAPTP301S mouse model of Alzheimer’s disease

Frontiers in Neuroscience

2024

Temporal alterations in white matter in an App knock-in mouse model of Alzheimer's disease.

eneuro

2024/2/1

Inflammasome signaling is dispensable for ß-amyloid-induced neuropathology in preclinical models of Alzheimer's disease.

Frontiers in Immunology

2024/1/29

Proteomic alterations in the brain and blood-brain barrier during brain Aβ accumulation in an APP knock-in mouse model of Alzheimer's disease.

Fluids and Barriers of the CNS

2023

A microtubule stabilizer ameliorates protein pathogenesis and neurodegeneration in mouse models of repetitive traumatic brain injury.

Science Translational Medicine

2023

Translocator protein is a marker of activated microglia in rodent models but not human neurodegenerative diseases.

Nature Communications

2023

A fully automated home cage for long-term continuous phenotyping of mouse cognition and behavior.

Cell Reports Methods

2023/7/24

Regional contributions of D-serine to Alzheimer's disease pathology in male App(NL-G-F/NL-G-F) mice.

Frontiers in Aging Neuroscience

2023

Prenatal stress aggravates age-dependent cognitive decline, insulin signaling dysfunction, and the pro-inflammatory response in the APP(NL-F/NL-F) mouse model of Alzheimer's …

Neurobiology of Disease

2023

Temporal progression of Alzheimer's disease in brains and intestines of transgenic mice

Neurobiology of aging

2019/9/1

Kumi Nagamoto-Combs
Kumi Nagamoto-Combs

H-Index: 10

Takashi Saito
Takashi Saito

H-Index: 31

Myelin dysfunction drives amyloid-β deposition in models of Alzheimer's disease.

Nature

2023

Alterations to parvalbumin-expressing interneuron function and associated network oscillations in the hippocampal - medial prefrontal cortex circuit during natural sleep in …

Neurobiology of Disease

2023

A tailored tetravalent peptide displays dual functions to inhibit amyloid β production and aggregation.

Communication Biology

2023

INPP5D modulates TREM2 loss-of-function phenotypes in a β-amyloidosis mouse model.

iScience

2023

Brain p3-Alcβ peptide restores neuronal viability impaired by Alzheimer's amyloid β-peptide.

EMBO Molecular Medicine

2023

Cerebral Aβ deposition precedes reduced cerebrospinal fluid and serum Aβ42/Aβ40 ratios in the AppNL-F/NL-F knock-in mouse model of Alzheimer's disease.

Alzheimer’s Research & Therapy

2023

Brain-derived major glycoproteins are possible biomarkers for altered metabolism of cerebrospinal fluid in neurological diseases.

International Journal of Molecular Sciences

2023

Reduced autophagy in aged trigeminal neurons causes amyloid β diffusion.

Journal of Dental Research

2023

See List of Professors in Takashi Saito University(Nagoya City University)

Co-Authors

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